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» » Experts have discovered a new cause of skin aging

Experts have discovered a new cause of skin aging

A team of experts found that the key enzyme in human skin decreases with age. This enzyme seems to play a crucial role in the maintenance of cell energy levels. With lower levels of this enzyme deteriorating health of the individual cells, and skin wrinkles and creases. Now that scientists have discovered the cause of skin aging, they are trying to figure out how to stop this process. The researchers hope that the new anti-aging agents, which will have a specific target in the form of the enzyme, allowing the skin to stay longer young and beautiful. Finally, the results can also be applied to other parts of the body, having the potential to stop the development of cancer and other diseases associated with aging.

As we age, the body's functioning mitochondria - the energy "batteries" - is getting worse in our cells, with the result that the amount of energy is reduced, and the levels of harmful free radicals increase. This process can easily be seen on a sagging skin and wrinkles of growth. A new study shows that with increasing age in human skin a decrease in the activity of a key enzyme found in the mitochondria. This enzyme is called mitochondrial Complex II maintains the energy levels in our cells. Having examined the levels of this enzyme in the skin samples of 27 people between the ages of 6 to 72 years, researchers found that the enzyme activity is significantly reduced with age.

According to scientists, they now have a specific biomarker, to which the new anti-aging treatments and cosmetic creams can be targeted to help prevent decline in energy levels in cells. Experts hope that their discovery will help develop a comprehensive anti-aging therapy, which can be adapted to different people, and will solve the problem of aging and in other parts of the body, such as in the brain. Thus, this work makes scientists one step closer to understanding how vital cellular structures may contribute to human aging.
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